Male Sexuality Facts

Facts, Theories, And Information on Male Sexuality: Male Sexual Problems

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Overcoming Premature Ejaculation

Premature ejaculation (PE) is a problem during which a man achieves orgasm in sexual intercourse - coitus - before he or his lover is ready. This may deprive her of sexual pleasure.

Sexologists Dr Masters & Virginia Johnston maintained that PE was a problem present when a man gets to the point of ejaculation prior to his lover in more than 51 percent of their sexual lovemaking. Presently the general description of early ejaculation is intercourse in which the male orgasms within 2 minutes after the time of penetration.

The hard reality is that research by Dr Kinsey even in the 1940s proved that 75% of men reach their  orgasm within three minutes - that is, three minutes after penetration - more than fifty percent of the time.

Rapid ejaculation is divided into several categories: primary PE occurs from the first time a male has intercourse; secondary PE begins somewhat later in a man’s life.

Quick ejaculation should be additionally categorized between “global PE”, implying a fast orgasm with all sexual partners, every time a man has sex, and “situational PE” – which is experienced only when a man is with a small number of specific lovers.

About half of men will almost certainly reach orgasm before sex is really underway. Since there can be considerable variation in how long sex lasts before men ejaculate, and because the pleasures from intercourse are so different, sexologists have now started to form a statistical way to define premature ejaculation.

Present data implies an average gap between intromission and ejaculation, also called the “intravaginal ejaculatory latency time” or IELT, of approximately six minutes in 18-29 year old human males.

When PE is characterized with reference to an IELT percentile of less than 2.5, then the expression “early ejaculation” might accurately be applied to an orgasm that takes place within two minutes of intromission.

However, it’s possible, if not probable, for men and their sexual partners with extremely short-loved ability to control ejaculation to be totally pleased with their sexual skills or to have no sense of their inadequate ejaculatory self-control.

Likewise men with more developed lovemaking ability may sometimes think about themselves as fast comers, experiencing unwanted premature release and needing help to slow things down even if an observer might see things differently.

The bodily process of orgasm consists of 2 connected mechanisms: emission and expulsion. Emission is the primary part of ejaculation. It requires the expelling of seminal fluid from the vas deferens and vesicles of the reproductive tract.

Emission is associated with a distinctive sensation which comes just before ejaculation. The prostate gland also secretes fluid into the back of the urethral tube.

Expulsion is the second part of ejaculation. It includes closure of the bladder sphincter, after which come the rhythmic contractions of the perineal and pelvic muscles and rhythmic relaxation and contraction of the exterior urethral sphincters. This mirrors the expulsion of fluid during orgasm produced by G spot stimulation in women.

We now believe that the neurotransmitter serotonin (5HT) has a central role in modulating ejaculation and orgasm. Several studies on rats have shown its inhibitory effect on the mechanism of ejaculation. Subsequently, it is perceived that low amounts of serotonin within the synaptic cleft in specific parts of the brain could trigger rapid ejaculation.

This concept is additionally given weight by the proven effectiveness of SSRIs (which enhance serotonin levels within the synapse), in curing premature ejaculation. Motor neurons of the sympathetic nervous system control the first, emission, phase of ejaculation, while expulsion of semen is controlled by autonomic motor nerve cells.

These motor nerve cells are situated in the thoracolumbar spinal cord and are activated in a highly co-ordinated manner when sensory stimulation reaches the reflex ejaculation trigger.

Particular parts of the brain tissue, especially the nucleus paragigantocellularis, have been proven to be concerned with control of ejaculation. Researchers have always believed there to be a genetic link in certain variants of PE: and in one research project, ninety-one percent of sexually active men who always came too fast had a close relation with lifelong premature ejaculation.

Different scientists have shown that men with a rapid climax have a more rapid neurological reaction in the pelvic muscles. A program of muscular workout routines can considerably enhance ejaculation control for men who have no control in lovemaking.

Many doctors feel PE is brought on by emotional issues like fear of failure and so forth. It may be that such men may benefit by taking anti-anxiety treatment such as or SSRIs such as paroxetine. These drugs can decrease ejaculation speed.

Some men prefer to use anesthetic creams which reduce physical sensations - either way, rapid ejaculation needs to be treated before any erectile dysfunction. (ED is found in almost 50% of males affected by premature ejaculation.)

To identify effective therapy for PE a analysis needs to be made utilizing the man’s entire sexual profile, searching for signs of change in IELT, and proof of poor ejaculatory control, emotional issues in man and sexual partners and stress in either the male or his relationship partner.

sexual pleasure

When deciding the most suitable remedy, it is vital for the physician to distinguish between PE as “a partner’s criticism” and PE as what is now known as a “syndrome”.

premature ejaculationThis male dysfunction may be categorized into lifelong and acquired.

Recently, a new classification was suggested based on controlled clinical stopwatch studies. Other terms have been suggested: natural variable PE and premature-like ejaculatory dysfunction.

Only PE which has existed for years showing ejaculation latency time of less than one and a half minutes should be regarded as a probable candidate for medical treatment as the first option, together with behavioral therapy.

Some categories of PE can be treated with emotional reassurance because rapid orgasm is a normal aspect of male sexual response. However, when things are more serious, drugs may be needed. And here is Dapoxetine!

This is a brief-acting SSRI developed to help men overcome premature ejaculation. Dapoxetine is the only drug with any approval for this use. Presently, it’s dispensed in a number of European nations, such as Finland. Priligy, the trade name, is said to be an effective treatment and seems safe.

Before the development of Priligy,  Clomipramine tended to be prescribed to lessen the symptoms of PE. Some other drugs used to cure PE include: Ultram, an American accepted oral painkiller for moderate pain. It’s similar to an opioid, works on the sensory receptors, but in addition is much like an anti-depressant in that it will increase levels of norepinephrine and serotonin.

The pain killer Tramadol has almost no negative effects, has low abuse potential, and will increase time to ejaculation more effectively than desensitizing lotions using Lidocaine.